antifade reagent Search Results


dapi  (MedChemExpress)
94
MedChemExpress dapi
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Dapi, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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dapi - by Bioz Stars, 2026-04
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dapi  (Vector Laboratories)
93
Vector Laboratories dapi
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Dapi, supplied by Vector Laboratories, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/dapi/product/Vector Laboratories
Average 93 stars, based on 1 article reviews
dapi - by Bioz Stars, 2026-04
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6 diamino 2 phenylindole  (Cell Signaling Technology Inc)
96
Cell Signaling Technology Inc 6 diamino 2 phenylindole
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
6 Diamino 2 Phenylindole, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/6 diamino 2 phenylindole/product/Cell Signaling Technology Inc
Average 96 stars, based on 1 article reviews
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prolong gold antifade  (Cell Signaling Technology Inc)
96
Cell Signaling Technology Inc prolong gold antifade
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Prolong Gold Antifade, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/prolong gold antifade/product/Cell Signaling Technology Inc
Average 96 stars, based on 1 article reviews
prolong gold antifade - by Bioz Stars, 2026-04
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prolong gold® antifade reagent  (Carl Zeiss)
90
Carl Zeiss prolong gold® antifade reagent
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Prolong Gold® Antifade Reagent, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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fluoro keeper antifade reagent  (Nacalai)
90
Nacalai fluoro keeper antifade reagent
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Fluoro Keeper Antifade Reagent, supplied by Nacalai, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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fluoro keeper antifade reagent - by Bioz Stars, 2026-04
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antifading reagent  (Citifluor Ltd)
90
Citifluor Ltd antifading reagent
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Antifading Reagent, supplied by Citifluor Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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antifade  (Citifluor Ltd)
90
Citifluor Ltd antifade
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Antifade, supplied by Citifluor Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/antifade/product/Citifluor Ltd
Average 90 stars, based on 1 article reviews
antifade - by Bioz Stars, 2026-04
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fluoro-keeper antifade reagent p7184  (Nacalai)
90
Nacalai fluoro-keeper antifade reagent p7184
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Fluoro Keeper Antifade Reagent P7184, supplied by Nacalai, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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antifade reagent zl-9556  (ZSGB Biotech)
90
ZSGB Biotech antifade reagent zl-9556
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Antifade Reagent Zl 9556, supplied by ZSGB Biotech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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prolongtmgold anti-fade reagent  (Servicebio Inc)
90
Servicebio Inc prolongtmgold anti-fade reagent
IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei <t>were</t> <t>counterstained</t> with <t>DAPI</t> (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.
Prolongtmgold Anti Fade Reagent, supplied by Servicebio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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x5 prolong gold antifade reagent  (Fisher Scientific)
90
Fisher Scientific x5 prolong gold antifade reagent

X5 Prolong Gold Antifade Reagent, supplied by Fisher Scientific, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei were counterstained with DAPI (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.

Journal: Journal of Inflammation Research

Article Title: Integrated Multi-Omics Analysis Reveals IRF1-Driven Microglial PANoptosis via ZBP1 in Spinal Cord Injury

doi: 10.2147/JIR.S574990

Figure Lengend Snippet: IRF1 drove Zbp1 transcription in microglia. ( A ) Heatmap showing TF activity scores (right) and fold-change expression (left) associated with PANoptosis activity in microglia. ( B ) UMAP visualization of transcriptional activity scores (top) and gene expression levels (bottom) of Nr1d1, Spi1, Myc, Irf1 , and Cebpd across all cell types. ( C ) Heatmap showing the results of Pearson’s correlation analysis between Nr1d1, Spi1, Myc, Irf1 , and Cebpd and PANoptosis activity scores. ( D ) Bar graph showing the inferred TF activity at bulk RNA-seq levels. ( E ) The IGV browser illustrating the potential regulatory relationship between NR1D1, SPI1, MYC, IRF1, and CEBPD and the Zbp1 promoter region. ( F ) UCSC Genome Browser showing a distinct IRF1 binding peak within the Zbp1 promoter region, based on public ChIP-seq data. ( G ) Immunofluorescence at 7 days after SCI showing nuclear translocation of IRF1 (red) in Iba1+ microglia (green); nuclei were counterstained with DAPI (blue). Scale bars, 20μm (n=4 rats per group). ( H ) WB analysis of IRF1 and ZBP1 protein expression of microglia in vitro. Vinculin was used as a loading control. *, p<0.05; ****, p<0.0001.

Article Snippet: Nuclei were counterstained with DAPI (HY-K1048, MedChemExpress).

Techniques: Activity Assay, Expressing, Gene Expression, RNA Sequencing, Binding Assay, ChIP-sequencing, Immunofluorescence, Translocation Assay, In Vitro, Control

Journal: EMBO Molecular Medicine

Article Title: A human monoclonal antibody bivalently binding two different epitopes in streptococcal M protein mediates immune function

doi: 10.15252/emmm.202216208

Figure Lengend Snippet:

Article Snippet: X5 Prolong gold antifade reagent , Fisher scientific , 11569306.

Techniques: Virus, Clinical Proteomics, Recombinant, Saline, Staining, Labeling, Plasmid Preparation, Microarray